Saturday, April 29, 2017

Optimize Your Mitochondrial Metabolism


We're now starting to realize that mitochondrial dysfunction is at the core of virtually all diseases, and support for nutritional ketosis is growing by leaps and bounds. 2016 was a breakthrough year for this kind of information.
For over 80 years, nutritional ketosis has been the standard of care for intractable seizures in children.
Now we're finding it can benefit a wide array of other diseases, including neurodegenerative diseases such as Alzheimer's and Parkinson's, obesity, diabetes, heart failure, heart disease, arthritis and more.     
One of the reasons it works so well is because it drives your inflammation down to very low levels. When inflammation disappears, your body can heal. It also takes the proverbial foot off the gas pedal of aging. My next book, "Fat for Fuel," scheduled for release in May 2017, will explain it all in detail.
Without this information, people will continue to die prematurely. At present, the cancer industry is focusing on the downstream effects of the problem, which is why the "war on cancer" has been such a miserable failure.
When you view cancer as a metabolic disease, you can actually target and manage the disease without creating systemic toxicity. You do this primarily by targeting the fuels the cancer cells use (primarily glucose).

Without the appropriate fuel, the cancer cells cannot grow and multiply. Six strategies that will help optimize your mitochondrial function include:

1. Peak Fasting and other types of fasting
2. Eating foods low in net carbs and protein and high in healthy fats
3. Optimize your iron levels by getting ferritin to 60 nanograms per milliliter (ng/mL)
4. Exercise
5. Reduce mitochondrial reactive oxygen species (ROS) production by avoiding food for at least three hours before bedtime
6. Get sensible sun exposure, as a majority of the energy your body needs to maintain systemic equilibrium comes from environmental infrared light exposure, and avoid light-emitting diode (LED) lighting    







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